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Zebrafish Models for Dyskeratosis Congenita Reveal Critical Roles of p53 Activation Contributing to Hematopoietic Defects through RNA Processing

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Zebrafish Models for Dyskeratosis Congenita Reveal Critical Roles of p53 Activation Contributing to Hematopoietic Defects through RNA Processing Empty Zebrafish Models for Dyskeratosis Congenita Reveal Critical Roles of p53 Activation Contributing to Hematopoietic Defects through RNA Processing

Post  Jon Moulton Mon Jan 30, 2012 4:56 pm

Zhang Y, Morimoto K, Danilova N, Zhang B, Lin S. Zebrafish Models for Dyskeratosis Congenita Reveal Critical Roles of p53 Activation Contributing to Hematopoietic Defects through RNA Processing. PLoS ONE. 2012;7(1):e30188. doi:10.1371/journal.pone.0030188
Jon Moulton
Jon Moulton

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