Defective Escape Behavior in DEAH-Box RNA Helicase Mutants Improved by Restoring Glycine Receptor Expression
Page 1 of 1
Defective Escape Behavior in DEAH-Box RNA Helicase Mutants Improved by Restoring Glycine Receptor Expression
Jon Moulton Mon Sep 16, 2013 11:21 am
Hirata H, Ogino K, Yamada K, Leacock S, Harvey RJ. Defective Escape Behavior in DEAH-Box RNA Helicase Mutants Improved by Restoring Glycine Receptor Expression. J Neurosci. 2013;33(37):14638-44. doi:10.1523/JNEUROSCI.1157-13.2013
Jon Moulton- Posts : 5721
Join date : 2010-03-12
Age : 61
Location : Philomath, Oregon, USA -
Similar topics» Duplicated gephyrin genes showing distinct tissue distribution and alternative splicing patterns mediate Moco biosynthesis, glycine receptor clustering and escape behavior in zebrafish
» The zebrafish maternal-effect gene mission impossible encodes the DEAH-box helicase Dhx16 and is essential for the expression of downstream endodermal genes
» Expression of CALR mutants causes mpl-dependent thrombocytosis in zebrafish
» Functional Consequences of the Postnatal Switch From Neonatal to Mutant Adult Glycine Receptor α1 Subunits in the Shaky Mouse Model of Startle Disease
» Whole-genome expression profile in zebrafish embryos after chronic exposure to morphine: identification of new genes associated with neuronal function and mu opioid receptor expression
» The zebrafish maternal-effect gene mission impossible encodes the DEAH-box helicase Dhx16 and is essential for the expression of downstream endodermal genes
» Expression of CALR mutants causes mpl-dependent thrombocytosis in zebrafish
» Functional Consequences of the Postnatal Switch From Neonatal to Mutant Adult Glycine Receptor α1 Subunits in the Shaky Mouse Model of Startle Disease
» Whole-genome expression profile in zebrafish embryos after chronic exposure to morphine: identification of new genes associated with neuronal function and mu opioid receptor expression
Page 1 of 1
Permissions in this forum:
You cannot reply to topics in this forum