Knockdown of prothymosin α leads to apoptosis and developmental defects in zebrafish embryos
Page 1 of 1
Knockdown of prothymosin α leads to apoptosis and developmental defects in zebrafish embryos
Jon Moulton Tue Sep 17, 2013 10:23 am
Emmanouilidou A, Karetsou Z, Tzima E, Kobayashi T, Papamarcaki T. Knockdown of prothymosin α leads to apoptosis and developmental defects in zebrafish embryos.. Biochem Cell Biol. 2013 Oct;91(5):325-32. doi: 10.1139/bcb-2012-0103. Epub 2013 Aug 13.
Last edited by Jon Moulton on Tue Sep 17, 2013 10:24 am; edited 1 time in total (Reason for editing : Add URL)
Jon Moulton- Posts : 5721
Join date : 2010-03-12
Age : 61
Location : Philomath, Oregon, USA -
Similar topics» IRE1α knockdown rescues tunicamycin-induced developmental defects and apoptosis in Xenopus laevis
» Ambra1 knockdown in zebrafish leads to incomplete development due to severe defects in organogenesis
» Splice blocking of zygotic sox31 leads to developmental arrest shortly after Mid-Blastula Transition and induces apoptosis in zebrafish
» NgAgo-based fabp11a gene knockdown causes eye developmental defects in zebrafish
» LRRK2 knockdown in zebrafish causes developmental defects, neuronal loss, and synuclein aggregation
» Ambra1 knockdown in zebrafish leads to incomplete development due to severe defects in organogenesis
» Splice blocking of zygotic sox31 leads to developmental arrest shortly after Mid-Blastula Transition and induces apoptosis in zebrafish
» NgAgo-based fabp11a gene knockdown causes eye developmental defects in zebrafish
» LRRK2 knockdown in zebrafish causes developmental defects, neuronal loss, and synuclein aggregation
Page 1 of 1
Permissions in this forum:
You cannot reply to topics in this forum|
|
|