Interaction of Infectious Spleen and Kidney Necrosis Virus ORF119L with PINCH Leads to Dominant-Negative Inhibition of ILK and Cardiovascular Defects in Zebrafish
Page 1 of 1
Interaction of Infectious Spleen and Kidney Necrosis Virus ORF119L with PINCH Leads to Dominant-Negative Inhibition of ILK and Cardiovascular Defects in Zebrafish
Jon Moulton Wed Oct 29, 2014 12:02 pm
Yuan J-M, He B-L, Yang L-Y, Guo C-J, Weng S-P, Li SC, He J-G. Interaction of Infectious Spleen and Kidney Necrosis Virus ORF119L with PINCH Leads to Dominant-Negative Inhibition of ILK and Cardiovascular Defects in Zebrafish. J Virol. 2014. [Epub ahead of print] doi:10.1128/JVI.01955-14
Jon Moulton- Posts : 5721
Join date : 2010-03-12
Age : 61
Location : Philomath, Oregon, USA -
Similar topics» The Viral TRAF Protein (ORF111L) from Infectious Spleen and Kidney Necrosis Virus Interacts with TRADD and Induces Caspase 8-mediated Apoptosis
» Deficiency of a transmembrane prolyl 4-hydroxylase in the zebrafish leads to basement membrane defects and compromised kidney function
» Ribosomal protein L11 mutation in zebrafish leads to haematopoietic and metabolic defects
» Knockdown of prothymosin α leads to apoptosis and developmental defects in zebrafish embryos
» Ambra1 knockdown in zebrafish leads to incomplete development due to severe defects in organogenesis
» Deficiency of a transmembrane prolyl 4-hydroxylase in the zebrafish leads to basement membrane defects and compromised kidney function
» Ribosomal protein L11 mutation in zebrafish leads to haematopoietic and metabolic defects
» Knockdown of prothymosin α leads to apoptosis and developmental defects in zebrafish embryos
» Ambra1 knockdown in zebrafish leads to incomplete development due to severe defects in organogenesis
Page 1 of 1
Permissions in this forum:
You cannot reply to topics in this forum